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9 minute and 45 second D2B time?

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Image credit: CantonRep.com

That’s the startling claim made in this report found at CantonRep.com.

A Plain Township emergency medical squad of Bill Meers, Craig Hamilton, David Schwitzgable, and Deputy Chief Don Snyder received Mercy Medical Center’s Emergency Chest Pain Center’s STEMI Cup award on Feb. 18. The squad was the first to receive the award.

The ST-segment elevation myocardial infarction award was given to the team for its role in the “door to balloon angioplasty” time of 9 minutes and 45 seconds. It is the fastest time ever reported for this procedure.

The award was presented during a continuing education unit meeting by Drs. Ahmed A. El Ghamry Sabe and Frank J. Kaeberlein, co-directors of Mercy’s ECPC.

Ranked among the top 10 in Ohio for overall cardiac services, Mercy also is recognized as the best in Stark County for overall cardiac care, cardiac surgery, cardiology, and PCI (angioplasty).

The EMS-to-balloon (E2B) time was not reported.

Fire/EMS blogosphere meet-up at EMS Today

3 comments

Heading to the EMS Today conference in Baltimore next week?

The “largest live gathering of EMS and Fire bloggers EVER” is scheduled to take place on Friday, March 5, 2010 at 8:00 p.m.

The event will be held at the Uno Chicago Grill at 201 East Pratt Street (the Pratt Street Pavilion – Zone B, Level 2) at the Inner Harbor which is only a block away from the Convention Center.

Click HERE for directions from Google.

The event is being sponsored by The George Washington University – Emergency Health Services Program, JEMS, and FireEMSBlogs.com.

Everyone is welcome! You don’t have to be a blogger.

The Happy Medic writes about it HERE. 999Medic writes about it HERE. The Fire Critic writes about it HERE. MedicCast writes about it HERE. FossilMedic writes about it HERE.

Image credit: Google
Image credit: Harborplace.com
Image credit: Harborplace.com
Image credit: Wikipedia

Transcutaneous pacing (TCP) with a Lifepak 12

5 comments
Image credit: Physio-Control

I discovered an interesting quirk about the Lifepak 12 the other day.

I’m sure many of you have been told (as I had been told) that the Lifepak 12 cannot perform TCP unless the limb lead electrodes are attached. There is a caveat to this (reference page 4-18 in the Operations Manual – this is a PDF file so “right-click” the link and select “save as”). If you are performing TCP in demand mode (even if you have it set well below the patient’s intrinsic rate and no pacing is being delivered) as soon as the monitor detects “leads off” the monitor will deliver TCP at a fixed rate until the leads are replaced or the pacer is turned off.

For example, say you have a patient with atrial fibrillation and a slow ventricular response of 50 BPM whose ventricular response occasionally drops down to 20 (with 3 – 6 second asystolic pauses during which time the patient loses consciousness and appears peri-arrest). You apply the combo-pads and set the demand pacer for 40 PPM @ 130 mA so that the patient’s heart rate cannot drop below 40 (assuming capture is achieved with 130 mA). The patient’s heart rate stays above 40 so no pacing is delivered.

At the hospital, the nurses (through no fault of their own) remove the ECG leads to switch the patient to their own Lifepak 20. What happens? Answer: The Lifepak 12 delivers fixed rate pacing at 40 PPM @ 130 mA through the combo-pads until the leads are replaced or the pacer is turned off. Not a big deal, just something to be aware of. This is not a device malfunction.

See also:

Transcutaneous pacing (TCP) – The problem of false capture

Using capnography to confirm capture with transcutaneous pacing (TCP)

58 year old male CC: Unconscious (Transcutaneous pacing failure in the setting of hyperkalemia)

Transcutaneous pacing (TCP) for asystole

Wisconsin EMS Tests Remote STEMI Project

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WLUK-TV Fox 11 News report found at JEMS.

((( VIDEO NO LONGER AVAILABLE )))

57 yom CC: Cardiac arrest

23 comments

Here’s another interesting case submitted by a reader who wishes to remain anonymous.

While we’re on the subject, I’d like to thank my readers for the interesting case studies that have been coming in!

This is what the Web 2.0 experience is all about! We’re learning a lot from each other and that’s the difference between editing a blog and writing a book.

EMS is called to a seizure patient. According to the patient’s girlfriend, he had been feeling weak and vomiting about an hour earlier. She states that he had a “short seizure” after which he was unresponsive. She didn’t call 9-1-1 right away because the patient has a history of seizures.

After the patient was unresponsivee for longer than usual she contacted 9-1-1. When asked by EMS personnel, she estimates that the patient had not been breathing for 3-5 minutes prior to EMS arrival. She did not attempt CPR.

The patient is a 57 year old male.

Past medical history: CVA, NIDDM, and seizures

Medications: Tylenol, Trileptol

Pulselessness was confirmed and chest compressions were initiated.

The combo-pads were attached.

Here is the initial rhythm (with CPR artifact).

It was confirmed as asystole a short time later.

CPR was continued. The airway was managed with an OPA and BVM. An 18 ga IV was established in the LAC and run w/o. Epinephrine and atropine were administered.

A rhythm change was noted on the monitor.

A weak carotid pulse was palpated although radial pulses were absent and a BP could not be auscultated.

The patient was relocated to the ambulance. The patient was intubated with an ETCO2 in the mid 50s.

A 12-lead ECG was captured.

The paramedics left the scene en route to a PCI-hospital.

At this point the patient had radial pulses but for some reason the paramedics were still unable to auscultate a blood pressure.

The rhythm continued to be irregular and polymorphic.

Another 12-lead ECG was captured.

Some significant asystolic pauses were noted on the monitor.

As the last 12-lead ECG was captured the patient lost pulses.

By now the ambulance was arriving at the PCI hospital. CPR was resumed. Another round of epinephrine was given and the patient re-gained pulses.

The ED staff identified the prehospital 12-lead ECGs as showing “STEMI”.

Do you agree? Why or why not?

Should it matter at this point? In other words, should the patient be cathed anyway (assuming the patient is stable enough for a cath)?

More on wide complex tachycardias

3 comments

From the ACLS Reference Textbook and Experienced Provider Manual (2003). Chapter 16: Stable Wide Complex Tachycardias:

“Electrical cardioversion is the definitive therapy for stable wide complex tachcyardia. It should be used immediately if the patient becomes unstable. It can avoid the potential complications resulting from the use of antiarrhythmic drugs, but its use may not always be feasible, desirable, or successful.

“Antiarrhythmic drugs require time to administer, and they have potential hypotensive, negative inotropic and proarrhythmic effects…”

“Most patients who develop ventricular rhythm disturbances have long-standing structural heart disease. Structural heart disease renders a patient at risk for adverse cardiac events during antiarrhythmic therapy. These adverse events include sudden arrhythmic death, unstable rhythms, stable tachycardias, and higher susceptibility to the proarrhythmic effects of antiarrhythmic agents.”

“[P]erhaps the most difficult [wide complex tachycardias] for the clinician to identify are supraventricular (SVT) or junctional tachycardias with aberrancy such as bundle branch blocks and intraventricular conduction delays…”

“Always obtain a 12-lead ECG before you provide any pharmacologic intervention for the stable patient. Repeat the 12-lead ECG after administering any antiarrhythmic and if the rhythm converts to a different rhythm. Review previous ECGs if they are available. A history of previous aberrant rhythms, accessory pathways, pre-existing bundle branch block, or rate-dependent bundle branch block suggests supraventricular aberrancy if the QRS morphology matches the QRS observed with the tachycardia.”

“The presence of a new-onset, stable, wide complex tachycardia, particularly one known to be present for more than an hour, inevitably raises the question, Is it ventricular tachycardia or supraventricular tachycardia with aberrancy? This ostensibly simple question merits detailed discussion.”

“Asked to inspect a series of wide-complex tachycardias and identify the rhythm as ventricular or supraventricular in origin, emergency and initial care providers respond incorrectly more than 50% of the time. Two “wrong answers,” or errors, are possible: the true rhythm is VT and the healthcare provider incorrectly assesses the rhythm as SVT with aberrancy, or the true rhythm is SVT with aberrancy and the provider assesses the rhythm as VT.

“Clearly, to minimize risk to the patient, the clinician must try to avoid making the error that would result in greatest potential harm to the patient. The guidelines have tried to prevent significant harm by introducing multiple places where the clinician must verify conclusions to detect diagnostic error. Specifically, the algorithm was constructed to reduce the risk of administration of verapamil to marginally stable patients with VT (if these patients are mistakenly diagnosed as having SVT with aberrancy); this mistake has been reported to be fatal.”

(Omitting a discussion about the difficulty in applying Wellens criteria and Brugada’s algorithm, the lack of validation, and the problem of interobserver agreement.)

“This debate has begun to acquire a patina of the esoteric because differentiating SVT with aberrancy from VT is not required in the ECC Guidelines 2000. To paraphrase the 1992-1999 guidelines, there are 2 rules for treating wide complex tachycardias of unknown type:

Rule 1: Treat all wide complex tachcyardias like VT.
Rule 2: Never forget Rule 1.”

I see nothing in the ECC Guidelines 2005 that contradicts this philosophy, and I don’t expect to see anything different in the ECC Guildelines 2010.

Amazing seat belt PSA

1 comment

h/t Pete R. from Star of Life Law

68 year old male CC: Chest Pain

44 comments

Here’s another great case submitted by Robert Bees.

EMS is called in the very early morning to a 68 year old male complaining of chest pain and shortness of breath.

On arrival, the patient is anxious and appears acutely ill. He is oriented to person, place, and time.

Skin is cool, pale, and diaphoretic.

The patient feels light-headed and admits to nausea but has not vomited.

Onset: While sleeping
Provoke: Nothing makes the pain better or worse
Quality: Poorly localized heavy pressure
Radiate: The pain does not radiate to the arms, back, neck or jaw
Severity: 8/10
Time: Patient states he occasionally “feels bad” but “not like this”

Past medical history: MI with stents x 2 years ago.

Medications: ASA, Lipitor, Tenormin, NTG

Patient states he took NTG x 1 prior to EMS arrival which made him “feel worse”

Vital signs:

Resp: 22 shallow
Pulse: 140
BP: 88/54
SpO2: 92 on RA

Breath sounds: rales – patient becomes very light-headed sitting up

The cardiac monitor is attached.

A 12-lead ECG is captured.

What now?

Peter Canning is the Man! (was: Paramedics Get Special Cardiac Training)

6 comments

Here’s a great story from WSFB-TV Channel 3 Eyewitness News (Hartford-New Haven) featuring our very own Peter Canning from the Street Watch: Notes of a Paramedic blog!

Watch as Peter calls in a “definite STEMI Alert” and activates the cath lab!

Click HERE for the best quality video without commercials.

Screen shots below.






Should you ever give a calcium channel blocker to a wide complex tachycardia?

34 comments

Some of you are probably wondering whether or not I’d ever condone giving a calcium channel blocker to a wide complex tachycardia in the field.

A recent case submitted by Robert Bees demonstrates a situation where I might consider it (or at least not criticize someone for considering it).

EMS is called to a 90 year old female with a chief complaint of difficulty breathing and weakness.

On arrival, the patient is alert and oriented to person, place, and time.

Vital signs:

Resp: 20 and non-labored
Pulse: 80 and regular
BP: 180/82
SpO2: 96 on RA

BGL: 121

Breath sounds are clear bilaterally.

Past medical history: HTN, asthma

Medications: prednisone, albuterol

The patient states that she used her inhaler prior to EMS arrival with no relief.

The patient denies chest discomfort. She states that she is not nauseated and she has not vomited.

The cardiac monitor was attached and a 12-lead ECG was captured.

The patient is placed on oxygen via NC @ 2 LPM and loaded for transport.

An IV is established in the left arm and 0.9% NS is run KVO.

Shortly after leaving the scene, a rhythm change is noted on the monitor.

Another 12-lead ECG is captured.

The patient became markedly more tired and went from alert to responsive to verbal stimuli.

What do these ECGs show?

The second 12-lead ECG shows a rhythm that is wide (QRS > 120 ms) and fast (HR > 100).

Is it ventricular tachycardia?

How do you know?

How would you treat this patient?

The Prehospital 12-Lead ECG blog receives its first hate mail

17 comments

 

I received an email today from a reader who ripped me a new one. Here’s the email with my replies interspersed.

To Whom This May Concern,

I stumbled upon this site while looking for additional information on poor r-wave progression. I happened to notice that in the wide-complex tachycardia section, there were some errors I would like to address.

So far so good! I generally appreciate it when someone points out my errors.

First and foremost, no one should treat unstable VT and unstable SVT the same, neither chemically, nor electrically. Unstable VT where losing a pulse is imminent, should be defibrillated where unstable SVT should always be synchronized cardioverted.

The 2005 AHA ECC guidelines classify tachycardias as either stable or unstable. Unstable tachycardias get cardioverted. It doesn’t matter if it’s a narrow complex tachycardia or a wide complex tachycardia. The caveat of course is that you should take steps to make sure it isn’t a compensatory tachycardia and attempt to identify reversible causes prior to cardioversion.

To my knowledge, there is nothing in the literature to substantiate unsynchronized cardioversion (defibrillation) for unstable regular wide complex tachycardias with a pulse.

In fact, having discussed this with various electrophysiologists, there’s no clinical reason why you shouldn’t synchronize the shock for a patient who is pulseless. Why not deliver the current at the optimal moment in the cardiac cycle? ICDs do it all the time, and they don’t check a pulse first.

I’ve researched this question extensively, and I’m left with the conclusion that VT without a pulse was thrown into the VF algorithm for simplicity, and because VT occasionally presents as ventricular flutter, during which time the computer may have difficulty differentiating between R-waves and T-waves. To prevent any delay (or mistakes by inexperienced rescuers) the pulseless VT algorithm calls for defibrillation.

If you have a better explanation, my ears are wide open.

Second, though amiodarone is a useful drug, many EMS departments do not wish to carry it because of its side effect profile, its questionable usefulness in upper chamber dysrhythmias, and its long half-life.

Sounds reasonable to me.

The literature supports lidocaine as being as effective as amiodarone for VT. Lidocaine is not as effective as calcium channel blockers in treating wide complex supraventricular irregular tachycardias however. 

But is it dangerous? Is it contraindicated? Obviously not, or L-A-P-B (lidocaine, adenosine, procainamide, bretylium) would never have been the ACLS algorithm for wide complex tachycardias of unknown or uncertain origin.

Third, it is academically dishonest and even perhaps illegal to directly quote from an article without a proper citation.

Is there a particular quote you’re referring to?

It is equally dishonest and professionally unethical to make criticisms of published materials using nothing more than mere opinion.

That’s just crazy talk!

Medical professionals criticize published material all the time, whether it be on blogs, editorials, letters to the editor, podcasts, webcasts, or online discussion forums and listservs.

The entire concept of publishing a paper is to put it out there for your peers to criticize. It’s one of the hallmarks of science.

Furthermore, I found your opinions unfounded, misguided, inaccurate, unkind, irresponsible, and lacking an academic foundation.

It would be extremely helpful to me to know what opinions of mine you found to be so irresponsible.

I advise you to use caution, sound judgement, graciousness, sound ethics and humility when publicly critiquing published materials.

Is this some kind of threat? If you’re going to question my ethics, I think I’m entitled to some examples.

It is a shame, but I cannot in good conscious take the contents of this website seriously. It contains more personal opinion than established fact. 

For example?

Furthermore, it appears that the author of the wide complex tachycardia section, who has a noticeably high opinion of himself, is more interested in attempting to sell people on his questionable knowledge than promote truths about electrocardiography.

By all means, enlighten me!

He publicly scathes an author of a wide-complex tachycardia article, but bases it on his own personal opinion, not academically sound research or practice.

Finally, a semi-specific criticism!

I assume you’re referring to my comments at the bottom of Differential diagnosis of wide complex tachycardias – Part I.

I will quote the relevant comments here:

[snip]

I recently discovered this article from the March 2006 issue of Emergency Medical Services. In it, the author states:

With the introduction of new pharmacological interventions that target specific areas of the cardiac conduction system, it has become increasingly important for EMS providers to make an accurate interpretation of an ECG. Though most paramedics have no difficulty distinguishing VT from narrow complex supraventricular tachycardia (SVT), some might fall victim to the “wide + fast = VT” trap when looking at SVT with aberrant conduction. Although VT and SVT with aberrant conduction look similar, they vary greatly in terms of origin, pathophysiology and treatment. Mislabeling dysrhythmias can have severe consequences. Improper identification of VT could place a patient in grave danger by delaying indicated pharmacological and electrical interventions.

Listen carefully.

“Wide and fast = VT” is not a trap! It’s a rule of thumb that exists to protect you and your patient!

The author continues:

A common aphorism among advanced practitioners is, “When in doubt whether a WCT is VT or SVT, treat patients as if they are experiencing VTs.” This stems from a statistic showing that approximately 80% of all WCTs are VT. Though this aphorism is generally a good rule of thumb, it is also important to acknowledge that one in five WCTs is not VT and therefore requires different treatment regimens. One must possess the proper diagnostic tools and knowledge to decide whether a WCT is VT or SVT with aberrant conduction. EMS providers should be able to differentiate VT and SVT with aberrant conduction with confidence and a high degree of certainty.

They do not require different treatment regimens!

Unstable SVT is treated the same as unstable VT! Anyone disagree?

It’s debatable whether or not a stable wide complex tachycardia should be treated in the field at all, but if you do reach for an antiarrhythmic, it better be one that works for SVT and VT (i.e., amiodarone or procainamide).

If you give a calcium channel blocker to a wide complex tachycardia without knowing with 100% certainty that it’s SVT with BBB (or aberrancy) you are a fool.

The article also contains outright errors. Here’s one of the most disturbing.

SVTs with aberrancy will produce either a right or left axis deviation. If the aberrancy is conducted in a RBBB pattern, right axis deviation will be present. If the aberrancy is conducted in a LBBB pattern, left axis deviation will be present. In almost all VT, the axis will be in the extreme right quadrant.

This is nonsense!

In the first place, RBBB and LBBB aberrancy can both show a normal axis. RBBB aberrancy in particular can show a normal axis, right axis deviation, or left axis deviation (bifascicular patterns).

Most cases of VT present with an other-than-extreme axis.

See what I mean about a little bit of knowledge being dangerous?

[snip]

So what it boils down to is I said, “This is nonsense!” and “See what I mean about a little bit of knowledge being dangerous?”

If you’re attacking me on style points, then point taken. I can be a bit brash sometimes. However, I stand by the principle behind my words. Using QRS morphology to differentiate between VT and SVT with aberrancy is a dangerous, error-prone skill.

Use it at your own peril.

Though I do not know the person’s background, it forces one to wonder whether this blogger is educated beyond what his own intelligence permits.

You are entitled to your opinion.

This has the potential to be a good website, but lacks credibility. I will avoid this site and advise others to do so until it can be based on a solid foundation of knowledge. Personal opinion does not cut it.

Thank you,
Matthew Paulus, BME, EMT-P, MS-S NP-S/Cardiopulmonary CNS-S

It’s a big Internet, Mr. Paulus. There are plenty of URLs to choose from. I’m sorry the Prehospital 12-lead ECG blog was not more to your satisfaction.

Have a wonderful day!

72 year old male CC: “Unknown problem” – Conclusion

9 comments

Here is the conclusion to 72 year old male CC: Unknown problem (man down)

Here was the initial 12-lead ECG.

 

Based on this ECG the lead paramedic called a "STEMI Alert" and transmitted the ECG to the receiving hospital.

The on-duty ED physician received the ECG and the paramedic's radio report.

The ED physician called up the patient's records on the computer system. It turned out that the patient had been to the hospital before.

There was a copy of a prehospital 12-lead ECG from March 2009 on file in the patient's chart.

 

Based on the similarities between this ECG and the ECG recorded on this call (and the fact that the presentation did not exactly scream ACS) the ED physician did not call the "Code STEMI" while EMS was still in the field.

It would prove to be the correct decision.

This is the 12-lead ECG that was captured on arrival.

 

You will note that this ECG is very similar to the prehospital 12-lead ECG captured back in March 2009. However, it's slightly different from the prehospital 12-lead ECG taken earlier that evening.

Go back up and look at the prehospital 12-lead ECG.

The frontal plane axis is off by about 15 degrees, the T-wave inversion in lead aVL is more subtle, and the R/S ratio in lead V2 is > 1.

Since these findings are not present in the 12-lead ECG taken on arrival at the hospital, it can probably be explained by lead placement.

Paramedics often project that attitude that skin prep and electrode placement are a low priority, but this case demonstrates why it's essential to quality patient care.

With careful lead placement and excellent data quality, the GE-Marquette 12SL interpretive algorithm does not give the ***ACUTE MI SUSPECTED*** message.

In addition, the ST-depression / inverted T-wave was a critical finding on the prehospital 12-lead ECG, because it suggested the possibility of a reciprocal change to the spurious ST-elevation in lead III.

The presence of the inverted T-wave on the "old" ECG made the ED physician take this finding with a grain of salt.

There's nothing wrong with having multiples sets of "critical eyes" looking at an ECG prior to calling in the cavalry, especially for a marginal ECG where it's questionable as to whether or not the "1 mm of ST-segment elevation in 2 or more anatomically contiguous leads" criterion is met.

That's how we minimize false positives, control health care costs, and do the right thing for the patient.

So we're 0 for 1 with our STEMI Alert protocol. However, on this particular day, the system demonstrated a hidden strength! The STEMI Alert allowed for a quick comparison to an "old" ECG.

Heart Safe Community PAD Program & STEMI Awareness Community Program Awards

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The International Association of Fire Chiefs is accepting nominations for the Heart Safe Community PAD Program & STEMI Awareness Community Program Awards.

Follow the IAFC on Twitter.

Become a fan on Facebook.

Note: You do not have to be a fire-based EMS system to win one of these awards! 

64 yom CC: Chest Pain

25 comments

Here’s another great case submitted by a faithful reader who wishes to remain anonymous.

The patient is a 64 year old male with a chief complaint of substernal chest pain.

Onset: Sudden onset at rest.
Provoke: Nothing makes the pain better or worse.
Quality: Patient describes the pain as “sharp”.
Radiate: Pain radiates to right arm.
Severity: Patient gives the pain a 15/10.
Time: 10 minutes prior to EMS activation.

Skin is cool, pale, and diaphoretic.

He denies shortness of breath. He admits to nausea but has not vomited.

Past medical history: CVA with right side deficit. IDDM with below knee amputation. Electric wheelchair bound.

Allergies: No known drug allergies

Medications: Vicodin

Vital signs:

Resp: 18
Pulse: very rapid, weak
BP: 103/81
SpO2: 99 on RA

BGL: 283

Breath sounds are clear bilaterally.

The cardiac monitor is attached.

 

A 12-lead ECG is captured.

 

And another.

 
What’s next? 
*** Update 02/12/2010 ***
A rhythm change is noted on the monitor.
An additional 12-lead ECG is captured.
 
And another.
 
Does this shed some light on the situation?

72 year old male CC: “Unknown problem”

22 comments

EMS is dispatched to a 72 year old male patient. Third party call. History of Parkinson's Disease. Patient is conscious. No further information.

On arrival, EMS finds a 72 year old Spanish-speaking male. Through an interpreter the lead paramedic determines that the patient became dizzy, fell down, and hit his head. A small hematoma is visible above the patient's right eye.

The patient is awake but somnolent. He is oriented to person, place, time, and event. The remainder of the neurological exam was normal.

Since the patient is not alert the crew applies manual C-spine stabilization and continues the exam.

The patient denies chest pain or shortness of breath.

Breath sounds are clear bilaterally.

The patient denies any significant medical history and states that he takes no medications.

Vital signs are assessed.

  • Resp: 18
  • Pulse: 80
  • BP: 104/70
  • SpO2: 98 on RA

The cardiac monitor is attached.

 
A 12-lead ECG is captured.
 
 
The lead paramedic notes that the arm leads are reversed. The problem is corrected and another 12-lead ECG is captured.
 
 
The black electrode is replaced and a third 12-lead ECG is captured.
 
 
What is your impression and what would you do next?
 

*** UPDATE ***

 

 
In the first graphic you can see that it's debatable as to whether or not 1 mm of ST-segment elevation is actually present in the 12-lead ECG when you use the TP segment as the baseline.

The first complex in lead III helps foster the perception, probably due to wandering baseline.

Compounding the illusion is the ST-depression in lead aVL! This is one of the first things I look for when considering the ECG diagnosis of acute inferior STEMI.

It's helped me pick up on dozens of subtle presentations!

 
In the second graphic I've blown up lead II so you can clearly see the PR-segment depression.

This is important for two reasons. First, it fools your eye into the thinking that ST-segment elevation is present. Secondly, it fools the GE-Marquette 12SL interpretive algorithm!

Having said that, I have respect for the GE-Marquette 12SL interpretive algorithm, and I'm certain it also picked up on the ST-depression in lead aVL.

Keep in mind that the ACC/AHA STEMI criteria is far from perfect. I've called STEMIs before with less than 1 mm of ST-segment elevation, specifically when ST-depression was present in lead aVL.

This case demonstrate that sometimes, the emergency department is exactly where a suspected acute STEMI patient (with a marginal ECG) belongs until the diagnosis can be confirmed through other means.

I'll be posting the conclusion to the case in the next couple of days.

See also:

72 year old male CC: Unknown problem (man down) – Conclusion

Modified Chest Leads (was: Modified Leads “On the Ice”)

3 comments

Occasionally I receive emails from readers who ask various questions or submit interesting ECGs which I sometimes use as case studies on the Prehospital 12-Lead ECG blog.

Back in November I received an email from a reader who wrote:

Hello! I’m currently in Antarctica on a medical support contract. I was issued a 3-lead Lifepak 10 for use at the field camp I’m staffing.

I’m wondering if you know of any way to manipulate/ alter this type of monitor so as to be able to gain V-lead views?

I kind of remember being shown some sort of trick years ago but, someone told me that it was probably demonstrated with a 4-lead monitor.

Any ideas? Many thanks in advance for your help!

Kindest Regards,
Ernie

I wrote him back with the following reply:

Hi, Ernie!

It’s easy. You just put the negative electrode in the position of the left shoulder and the positive electrode in the position of V1 (for example) and the result is MCL-1 (modified chest lead 1) which mimics V1. If you place the positive electrode in the position of V2 it’s MCL-2, and so on.

The fact that it’s a 3-lead is irrelevant because even a 4-lead is technically still a 3-lead. By designating the 4th electrode as the “ground lead” you can view all 3 leads (I, II, and III) at the same time. With a 3-lead you can only view one at a time because the 3rd lead defaults to the ground lead. For example, if you’re viewing lead II with a 3-lead it’s white (negative) to red (positive) and the black becomes the ground lead. If you’re viewing lead III then it’s black (negative) to red (positive) and the white becomes the ground lead.

One final suggestion. If memory serves with a LP10 you have to hold down a button (maybe the print button) continuously until a DIAG appears in the corner of the monitor screen which puts you in a diagnostic frequency response (where the low frequency/high pass filter goes from 1 to 0.05 Hz) so you can record accurate ST-segments. I hope this helps! Thanks for the question. Have a great day! Stay warm! Send pictures too if you get a chance! I’ll put you on my blog! :)

Tom

Well, I just heard back from him yesterday:

Tom,

Not sure if you remember me; I was the guy working in Antarctica that got in touch with you a few months ago to learn how to gain modified chest leads with the LP-10.

Well, I didn’t forget about you. I got home to California a couple of weeks ago, and have been getting caught up on email ever since! As promised, I’ve attached some pictures. You have my full permission to use any of the pics on your blog; I won’t be offended if you don’t use any though – I understand that it would be an odd entry for a blog dedicated to 12 lead education!

While I didn’t have to connect any patients to the LP-10, I was happy that I knew how to gain MCL’s 1 thru 6 if need be. Thanks again for your prompt and informative reply back in November.

Let me know if you have any questions about the pictures, or anything else.

Take Care,
Ernie

Here are the pictures he sent.

Here’s Ernie after a well-deserved haircut!

Thanks, Ernie! I’m glad I could be of assistance! Welcome home.

EMS Today – The JEMS Conference & Exhibition – March 2-6, 2010 – Baltimore Convention Center

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I am now registered to attend EMS Today. Who else is going?

http://c.brightcove.com/services/viewer/federated_f9/22978752001?isVid=1&publisherID=22899959001

*** Update 02/05/2010 ***

I found out yesterday that Justin Schorr (The Happy Medic) and Mark Glencorse (999 Medic) will be attending the EMS Today Conference through the Chronicles of EMS project!

The Happy Medic writes about it HERE.

999 Medic writes about it HERE.

Heart Health Awareness video from Denver Health

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A faithful reader (and movie star) brought an interesting PSA to my attention.

It’s a Heart Health Awareness video from Denver Health.

The video is interesting because it shows a heart attack and associated cardiac arrest from a patient’s perspective, including all phases of care.

First, the patient is struggling as he walks down the street.

After the patient collapses he regains consciousness in the back of an ambulance.

The patient goes into VF and is shocked. He regains consciousness in the emergency department.

He codes again and regains consciousness after successful PCI in the cath lab.

Finally, we see the cardiologist responsible for the follow-up care.

This video was a good reminder that things look very different from the patient’s perspective! Often times our patients are confused and scared. We can do a lot to reassure and encourage them.

Here’s the complete video clip.



Lawmakers shown first-hand how STEMI heart attacks are treated in Iowa

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Representative Ako Abdul Samad – Photo credit: WHO-TV

WHO-TV is reporting this morning that law-makers in Iowa were given a demonstration as to how regionalized STEMI care might work.

“Representative Ako Abdul Samad didn’t really have a heart attack, he was just playing the role.

For Monday, he was a victim of a STEMI — an ST-elevated myocardial infarction — a common type of heart attack that, if identified early and treated properly, can be subdued.

Des Moines Mercy is equipped with a PCI center, which can open blocked arteries but not every hospital in Iowa is, and getting word to them is the reason for involving legislators.

It’s important for legislators to understand that there are opportunities to create a coordinated system where we get people to where they need to go,” says Dr. Tom Evans, president of the Iowa Healthcare Collaborative.”

Such a system would identify heart attacks with the same equipment, treat them with the same medicine and send STEMI victims immediately to hospitals with PCI centers.”

A press release from IowaPolitics.com suggests that the simulation was conducted by the Iowa chapter of AHA Mission: Lifeline.

You can follow AHA Iowa on Twitter by clicking HERE.

*** Update 02/03/2010 ***

Video clip found at JEMS.com.

http://eplayer.clipsyndicate.com/cs_api/get_swf/3/&pl_id=8178&page_count=5&windows=1&va_id=1286031&show_title=0&auto_start=0&auto_next=0

Walt Disney World AED Program (was: Pathetic response by Disney!)

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In my previous post “Inappropriate or inappropriate ICD shocks – Part III” I talked about my experience contacting the “Big 3″ implantable medical device companies (Boston Scientific, Medtronic, and St. Jude Medical).

Here’s what I wrote:

This was nothing like dealing with the banking, cell phone, or computer industries!

  • There were few tele-prompt menus keeping you away from live help.
  • In each case, the person who answered the phone was friendly and courteous.
  • If I needed to be transferred, I wasn’t randomly disconnected.
  • The person who transferred the call stayed on the line and introduced me.
  • The technical experts knew the products intimately.
  • Questions were answered confidently.
  • They followed up with emails.

In short, the implantable medical device industry understands customer service!

It was like dealing with Disney World!

I lied. It was significantly better than dealing with Disney World.

To give you some background, I come from what you might call a “Disney family”. My parents are Disney Vacation Club members and it’s not uncommon for us to visit Disney World several times a year.

Speaking for myself, I love Disney World! If you haven’t been there in the past 10 years, you really need to go again and check out Soarin’, Expedition Everest, Mickey’s PhilharMagic, Festival of the Lion King, and Toy Story Mania! It’s a guaranteed good time.

In case you didn’t know, Disney is world renowned for customer service. It’s not rocket science really. They just treat people nicely. Sadly, that’s a forgotten concept in our hustle and bustle world. I think we’re all quite beaten down and our expectations have become ridiculously low, thanks to fast food and mega-chain stores.

When we’re treated the right way, it’s a pleasant surprise!

But not with Disney. We’ve come to expect outstanding customer service from the Mouse. It’s not cheap to visit Disney World, but we’re willing to pay it because they deliver the “magic”.

Which is why I’m really disappointed with the response I got from Disney World with regard to my recent request for information about their AED program.

My parents invited the entire family down to Disney World the week before Christmas. We stayed at Disney’s Boardwalk Inn, which is my parents’ favorite Disney resort. It’s walking distance from EPCOT and you can watch the fireworks outside your window at night.

Here are some pictures of the hotel.

I have no complaints about our Disney vacation, because Disney pulls out all the stops at Christmas. Everything was amazing and we had a wonderful time!

However, while I was at Disney’s Boardwalk Inn, I noticed that there were AEDs at the hotel.

One of them was on the ground floor, right outside of Muscles & Bustles Health Club. I especially liked that there was a phone next to the AED! Great idea.

Another was on the 4th floor right outside the elevators.

Before I go any further, I’d like to say that I commend Disney for placing AEDs at their hotels and throughout the parks.

When I got home, I went to Disney.com and looked for information about their AED program. Finding none, I used the generic contact form at the website to identify myself as the Editor of the Prehospital 12-Lead ECG blog and ask if there was someone I could talk to about the AED program.

A couple of weeks later, I finally received a response from someone at Disney Publishing Worldwide who requested my contact info. I provided it and as of today I have not heard back from them.

Very disappointing!

Perhaps Disney could learn a thing or two from the Implantable Medical Device industry.