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Right ventricular infarction – Part III

14 comments

Let's take a look at another case.

This was one of the first ECGs ever transmitted to my local receiving hospital on the Lifenet Receiving Station. It was definitely the first STEMI.

The data quality of the first 12 lead ECG wasn't the greatest. This is the second ECG, with lead V4 in the position of V4R.

Unfortunately, I can't seem to locate the details of this case. All I remember is that it was a male patient with chest pain.

This is an interesting ECG for several reasons.

There is ST segment elevation in leads II, III, and aVF which suggests acute inferior STEMI. You can also make out ST segment elevation in leads V5 and V6.

But where are the reciprocal changes? Normally we'd expect to see something in leads I and aVL. In this case, we don't even have so much as a flattening of the ST segment.

Very unusual indeed!

The only places I can see ECG changes that could be construed as reciprocal changes are in leads aVR and V1.

Is this a STEMI? I wouldn't blame you if you gave serious consideration to another diagnosis like pericarditis.

It is a STEMI.

Let's look at lead V4R. Do you see ST segment elevation?

No.

In fact, there appears to be about 1 mm of ST segment depression.

Is there right ventricular involvement?

No.

The culprit artery isn't even the RCA. It's the circumflex (LCX).

Take a look at the image to the right from an editorial in the New England Journal of Medicine by HJ Wellens.

You will note that lead V4R in this case looks almost identical to the third example, which indicates occlusion of the circumflex artery.

When I contacted the director of cardiovascular services at the hospital, he confirmed that the circumflex was 100% occluded.

If you remember your coronary anatomy from Part I, it's the right coronary artery (RCA) that typically supplies the right atrium and right ventricle before reaching the inferior wall of the left ventricle.

In a minority of patients, the circumflex (LCX) supplies the inferior wall of the left ventricle. Occlusion of this artery generally does not threaten the right ventricle.

So what have we learned? Is it always necessary to check the right sided precordial leads in the setting of acute inferior STEMI? Or at least lead V4R? It certainly isn't going to hurt. I won't discourage it.

Consider this comment left by Shaggy in Part II.

I work in a busy ED and one day the medics brought in a hypotenisive patient with an inferior wall MI on their 12 lead. I asked the attending if she wanted me to do a 12 lead with V4R. Her answer which I heard from others was if it is inferior and hypotensive, consider it right sided and treat as such. However, after reading this post, I see the importance of checking the right side on a normotensive patient with an inferior MI. I am glad you are around. I just wish I didn't have to keep reviewing your posts.

I tend to agree with the attending. I would simply include patients who are technically normotensive but on the low side of "normal" especially if they are bradycardic or "shocky" in appearance!

SoCal Medic alluded to another trick in a comment he left for Part I.

I have been taught two different ways, the first by obtaining V4R and evaluating that for ST Segment changes and the second by comparing Lead II to Lead III.

You will notice that in Part II, the ST segment elevation in lead III is > than the ST segment elevation in lead II. An examination of lead V4R confirms right ventricular involvement.

In this case, the ST segment elevation in lead II is > than the ST segment elevation in lead III. An examination of lead V4R confirms that there is not right ventricular involvement.

Is it really that simple? Actually, it is.

Consider this table from Eskola et al. How to Use ECG for Decision Support in the Catheterization Laboratory – Cases With Inferior ST Elevation Myocardial Infarction. Journal of Electrocardiography Vol 37 No. 4 October 2004.

See also:

Right ventricular infarction – Part I

Right ventricular infarction – Part II

Right ventricular infarction – Part III

Additional resources:

From the March 2008 issue of EMS Magazine:

Recognition and Treatment of Right Ventricular Myocardial Infarction
by Gene Gandy

14 Comments

  1. Tazambo says

    Hi Tom,I just noticed that your profile pictured changed again, that’s 2 new ones in as many weeks, isn’t it?I should really post my photo, it nice to see who’s actually posting.RegardsDave

    on February 17, 2009 @ 3:30 am.
  2. Tom B says

    Hi Dave,It’s 3 if you include the South Park icon I created at South Park Studio.I took it down after a couple of hours because it didn’t seem professional enough. :) I had the most recent picture taken yesterday for an interview I did with the EP Lab Digest.It should be published in the March 2009 issue.Tom

    on February 17, 2009 @ 7:00 am.
  3. qatardad says

    The simple rule I always teach is this:1. Any suprapubic complaint gets a routine 12 lead.2. Any inferior MI gets a V4R3. Any inferior MI gets fluid for Frank-Starling priming and we hold off on NTG and MS, or do a careful trial after a bolus (as you described) but obviously go ahead with ASA. Nice blog, BTW. I run paramedicine.com.Marc

    on April 1, 2009 @ 9:05 am.
  4. Anonymous says

    Hi Tom.Do you give boulus fluid, and hold in with nitro, to all pt with right ventricular infarction, even if they are normotensive ?Sorry for my bad english. /Jenny. Sweden.

    on September 3, 2009 @ 10:56 pm.
  5. Tom B says

    qatardad (Marc) – Thanks for the comment! Sorry I missed it.Tom

    on September 3, 2009 @ 11:06 pm.
  6. Tom B says

    Jenny – If the BP is on the low side of normal with inferior STEMI then I go ahead with a fluid bolus.At the very least I obtain IV access before the first trial dose of NTG!Tom

    on September 3, 2009 @ 11:07 pm.
  7. Anonymous says

    How about posterior involment in RV infarction ? Should ST depression in V1-v3(v4) be a "heads up" for RV involment as well in as inferior infarctions? The RCA supports the posterior wall to, doesnt it ?Thanks again for a really nice blogg. Keep it up./Rookie

    on September 3, 2009 @ 11:53 pm.
  8. Tom B says

    Rookie – The RCA often supplies the posterior wall through the posterior descending artery, but that's a distal branch of the RCA.It's a proximal occlusion of the RCA we're concerned with! I always look for the tell-tale ST segment depression in the right precordial leads with inferior ST elevation, but it's not my main tip-off that I'm dealing with RVI! I still feel the best evidence is STE in lead III > STE in lead II, although STE in V4R is also part of the puzzle (and let's not forget the physical exam).Tom

    on September 4, 2009 @ 7:40 am.
  9. Hillis says

    I know the article was posted around year but just i'd like to thanke you Tom for your great work .. I should admit in each case am learning and revising alot of information that i've unfortunately missed and still learning !!. The interpretation of ECG is so tricky !!Thanke you so much

    on January 13, 2010 @ 6:01 am.
  10. Tom B says

    My pleasure, Dr. Hillis! Thank you for reading my blog!

    Tom

    on January 2, 2011 @ 9:45 am.
  11. muataz says

    thank u Tom its really informative

    on January 5, 2011 @ 6:36 pm.

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Continuing the Discussion

  1. Right ventricular infarction – Part I – Prehospital 12-Lead ECG linked to this post

    [...] Right ventricular infarction – Part III Share and Enjoy: [...]

    on January 2, 2011 @ 9:41 am.
  2. Right ventricular infarction – Part II – Prehospital 12-Lead ECG linked to this post

    [...] Right ventricular infarction – Part III Share and Enjoy: [...]

    on January 2, 2011 @ 9:42 am.
  3. 49 year old male CC: Chest pain – Conclusion – Prehospital 12-Lead ECG linked to this post

    [...] I was wondering if anyone would notice that I’ve shown this ECG before. It’s from my 3-part tutorial on right ventricular infarction. Specifically it’s from Part III. [...]

    on April 28, 2011 @ 8:57 am.